Quinoline-3-carboxamides modulate primary T cell-dependent B cell responses but do not inhibit functional immunity.
Källberg E, Ivars F, Leanderson T.
Scand J Immunol · research support, non-u.s. gov't; journal article · DOI 10.1111/sji.12152 PUBLIKATIONSREGISTRET
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Immunol Lett · research support, non-u.s. gov't; journal article · DOI 10.1016/j.imlet.2004.07.004Negative regulation of FcepsilonRI signaling by FcgammaRII costimulation in human blood basophils.
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J Allergy Clin Immunol · research support, u.s. gov't, p.h.s.; research support, non-u.s. gov't; journal article · DOI 10.1067/mai.2000.107931A monoclonal autoantibody that promotes central nervous system remyelination in a model of multiple sclerosis is a natural autoantibody encoded by germline immunoglobulin genes.
Miller DJ, Rodriguez M.
J Immunol · research support, u.s. gov't, p.h.s.; research support, non-u.s. gov't; journal article · DOI 10.4049/jimmunol.154.5.2460Murine lupus anti-DNA antibodies cross-react with the hapten (4-hydroxy-5-iodo-3-nitrophenyl)acetyl, but immunization-induced anti-DNA antibodies do not.
Zouali M, Migliorini P, Stollar DB.
Eur J Immunol · research support, u.s. gov't, p.h.s.; journal article · DOI 10.1002/eji.1830170412Fine-specificity of the contact sensitivity to 4-hydroxy-3-nitrophenyl acetyl (NP).
Matoso-Ferreira A, Mäkelä O.
J Immunol · research support, non-u.s. gov't; journal article · DOI 10.4049/jimmunol.130.1.97Chemical basis for diversity in antibody specificity analysed by hapten binding to monoclonal anti-4-hydroxy-3-nitrophenacetyl (NP) immunoglobulins.
Mariuzza R, Strand M.
Mol Immunol · research support, u.s. gov't, p.h.s.; journal article · DOI 10.1016/0161-5890(81)90006-7Hapten-specific T cell response to 4-hydroxy-3-nitrophenyl acetyl. X. Characterization of distinct T cell subsets mediating cutaneous sensitivity responses.
Sunday ME, Dorf ME.
J Immunol · research support, u.s. gov't, p.h.s.; journal article · DOI 10.4049/jimmunol.127.2.766Hapten-specific T cell responses to 4-hydroxy-3-nitrophenyl acetyl. VI. Evidence for different T cell receptors in cells that mediate H-21-restricted and H-2D-restricted cutaneous sensitivity responses.
Sunday ME, Benacerraf B, Dorf ME.
J Exp Med · research support, u.s. gov't, p.h.s.; research support, non-u.s. gov't; research-article; journal article · DOI 10.1084/jem.152.6.1554Hapten-specific T-cell responses to 4-hydroxy-3-nitrophenyl acetyl. I. Genetic control of delayed-type hypersensitivity by VH and I-A-region genes.
Weinberger JZ, Greene MI, Benacerraf B, Dorf ME.
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