Glucosylceramide in humans.
Messner MC, Cabot MC.
Adv Exp Med Biol · research support, non-u.s. gov't; review; journal article; research support, n.i.h., extramural · DOI 10.1007/978-1-4419-6741-1_11 PUBLICATION REGISTRY
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5 hitsA possible mechanism underlying the ceramide deficiency in atopic dermatitis: expression of a deacylase enzyme that cleaves the N-acyl linkage of sphingomyelin and glucosylceramide.
Imokawa G.
J Dermatol Sci · review; journal article · DOI 10.1016/j.jdermsci.2009.04.006Abnormal expression of the novel epidermal enzyme, glucosylceramide deacylase, and the accumulation of its enzymatic reaction product, glucosylsphingosine, in the skin of patients with atopic dermatitis.
Ishibashi M, Arikawa J, Okamoto R, Kawashima M, Takagi Y, Ohguchi K, Imokawa G.
Lab Invest · research support, non-u.s. gov't; journal article · DOI 10.1097/01.lab.0000059931.66821.92High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis.
Hara J, Higuchi K, Okamoto R, Kawashima M, Imokawa G.
J Invest Dermatol · journal article · DOI 10.1046/j.1523-1747.2000.00072.xThe skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide.
Higuchi K, Hara J, Okamoto R, Kawashima M, Imokawa G.
Biochem J · research support, non-u.s. gov't; research-article; journal article · DOI 10.1042/bj3500747